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The below is taken from Dr. Rowen's website and is a published journal article on how direct IV ozone seemingly cured several Ebola cases. If it works for Ebola, it will work for many other viruses!

In October 2014, I traveled to Sierra Leone with my colleague Howard Robins to train local health professionals to treat Ebola with ozone therapy. Training went great. We hit a snag. While at the Sierra Leone Ebola treatment center outside the capital, a call came in from the Ministry of Health halting the ozone project. Patients were forbidden to receive ozone, and the staff as well. The staff did continue with our training fearing for their lives otherwise. Patients were denied and left to die.

As "luck" would have it, several health providers, who were on the front line, subsequently did come down with Ebola. Additionally we know of three additional doctors who contracted the disease. Of these three, two outright refused ozone therapy and quickly and miserably died. These made international news. The third was trained by me and requested ozone and was REFUSED. He also, sadly, died miserably. The remaining 4 managed to get the therapy and all 4 responded nearly instantly, totally recovered within a few days and had no complications. The government announced in the world news that one military physician did recover, and seemed to take credit. However, the government omitted the fact that he received ozone therapy.

We have just published the results of our 4 cases in the African Journal of Infectious Diseases, and I am providing it here for you to read and enjoy. A button below it can provide you with a download to the original PDF of the article.

Please also know that a fifth person, the female consort of one of the senior doctors, who refused ozone and died, had encouraged him to get ozone. She was placed under armed guard quarantine at her home and could not exit to get ozone prophylaxis. Fearing for her life, having had intimate exposure, she scaled a razor wire fence in the middle of the night, shredding her skin, to evade the guards. She was able to get to ozone and developed no symptoms. This is a story made for Hollywood. I'll avoid the rest of the ramifications of what happened for now. But the good news is that Ebola appears to be easily cured and for less than 10 USD! I have.

Rowen et al., Afr. J. Infect. Dis. (2016) 10 (1): 49– 54


1Robert Jay Rowen, MD, 2Howard Robins, DPM, 3Kojo Carew, MD, 4Michael Morlai Kamara, MD, 5Mohamed Ibrahim Jalloh.
1) 2200 County Center Dr. Ste C, Santa Rosa, California, USA 95403. Lead author and correspondence author, 2) 200 West 57th Street #807, New York, NY 10019. ,3) c/o Blue Shield Hospital, 27 Ascension Town Road, Freetown, Sierra Leone. 4) 66 Mayenkinneh Hills, Calaba Town, Freetown, Sierra Leone, 5) 31 Nelson Lane, Tengbeh Town, Freetown, Sierra Leone, West Africa.


Background: Ebola Virus Disease (EVD) has ravaged three countries in West Africa. The mortality rate is extremely high, and it is perceived not only as threat to all of Africa but to the entire world. There is no known treatment to date other than administration of convalescent blood or experimental monoclonal antibodies, which both often fail. Ozone therapy (OT) has been in clinical use for decades and has been found to have physiological effects, which should directly inactivate the virus itself, as well as modulate its damaging effects. We present the scientific background and the possibility of ozone therapy as a cure or prevention for EVD in five consecutive patients.

Materials and Methods: Ozone therapy administration by a combination of direct intravenous gas administration, rectal gas administration and ozonized water was administered to three patients with known acute EVD, one with apparent acute infection, and one case of extremely high risk. Treatment was carried out for up to ten days despite fast total remission of symptoms. Vitamin C and glutathione supporting supplements were administered.

Results: Four symptomatic patients, three with test positive EVD confirmation and one (who suffered Ebola contaminated needle stick contamination three days earlier) without lab confirmation all remitted symptoms within 2-4 days and fully recovered. All four ill cases had an immediate recovery course upon initiation of therapy. The single case of non-symptomatic high-risk exposure treated preventively did not develop symptoms.

Conclusion: Ebola virus may have a very narrow window of redox infectivity capacity, which can be easily exploited with OT. OT may be a useful modality in EVD and other viral diseases and should be immediately studied to save lives that might otherwise be lost.

Key words: Ebola, Hemorrhagic Fever, ozone therapy, oxidation, virus, antiviral therapy

A Filovirus of several strains, commonly known as Ebola, causes hemorrhagic fever (EVD). Incubation ranges 2-21+ days. Ebola virus enters dendritic cells shutting down their immune system alerting alarms. While unchecked, it replicates wildly, infecting and damaging critical organs, inclusive of liver and kidneys. The cells explode releasing new viruses and debris. This can result in a cytokine storm (Tisoncik et. al., 2012), characterized by massive capillary leakage and tissue destruction. Then, the immune system may do more harm than good (Amarasinghe, 2014). Mortality is extremely high (60% in the current Zaire species outbreak).

WHO reported that the current Ebola epidemic ravaging parts of West Africa is the most severe acute public health emergency seen in modern times (Watson, 2014). To date, there is no known proven effective treatment” (Choi and Croyle, 2013). A recent JAMA editorial proclaimed need for “utmost urgency” of fast tracking promising vaccines (Giesbert, 2015). Effective treatment is emergently needed.

Ozone is an oxygen allotrope, O3, created by solar UV radiation and lightning. It’s the strongest naturally occurring oxidant. Scripps Institute reported that ozone is actually generated by immune cells (Bablor, et. al., 2003) as part of its armamentarium of oxidants, which can be hurled against pathogens. Other immune system generated anti-infection oxidants include singlet oxygen, H2O2, NO, and NaOCl.

Ozone kills bacteria in easily reachable concentrations nearly instantly (Leusink and Kraft, url) and some 100 times faster than chlorine containing disinfectants (decontaminants) (Oregon State Univ., 2011).

Nikola Tesla patented the first commercial medical ozone generator in 1896. In World War I, German physicians disinfected wounds with medical OT. German physicians soon discovered that ozone application to various body fluids or cavities resulted in additional benefits, including enhanced circulation, oxygen delivery, and faster healing.

OT has been continuously used for nearly 100 years, especially in Europe for a variety of infectious, immunological and circulatory conditions. Velio Bocci, MD of Italy investigated OT’s immune effects. He published results first in a series of studies (over 175) in peer reviewed medical journals, now found succinctly in his book “Ozone, A New Medical Drug” (2011), which details ozone’s many mechanisms to help many medical conditions including: 1) immune system modulation balancing its inflammatory/anti-inflammatory cytokines, 2) increase in production of RBC oxygen releasing 2,3 DGP (also reported by Viebahn-Hansler, 2003), and improved rheology properties of blood (increased RBC flexibility) 3) elevation of key anti-oxidant enzymes such as SOD, and increased glutathione. Cuban ozone researchers have independently verified these findings (Menendez, et. al., 2008).

Medical OT induces vasoprotective prostacyclin production (Schulz, et. al., 2012). OT increases muscle (Clavo, et. al., 2003) and tumor oxygenation (Clavo, et. al., 2004) in humans studied by direct polarographic electrode measurement. Additionally, ozone protected against hepatic ischemia free radical reperfusion injury (Perralta, et. al., 1999).

Cuban researchers found that OT preconditioning inhibits TNF-alpha production during endotoxic shock. In addition OT exerts influence on the antioxidant-prooxidant balance for preservation of cell redox state by stimulating endogenous antioxidant systems (Zamora, et. al., 2005).

Many viruses require reduced sulfhydryl groups on their lipid envelope glycoproteins for cell entry. Mirazimi’s group speculated on the richness of disulfide bonds in viral glycoproteins as a factor for infectivity. Studying Cytomegalovirus, (CMV) they found the virus requires sulfhydryl groups to infect cells (Mirazmi, et. al., 1999). If the thiol groups were oxidized, CMV lost infectivity. When thiols were chemically re-reduced (by dithiothreitol), the virus regained 65% infectivity. Reflecting on the reduction of “critical” disulfide bonds for vaccinia virus cellular entry, Markovic, et. al., (2004) found that protein disulfide isomerase inhibitors limited HIV-1 entry into T cells.

Ozone inactivates many viruses including polio, Norwalk, coliophage MS2, hepatitis A and others (Kekez and Sattar, 1997; Shin and Sobsey, 2003; Herbold, et. al., 1989; Emerson, et. al., 1982).

Ebola appears no different. Studies by Sanders et al. have been able to determine the disulfide-bond map of the Ebola glycoprotein and, as a result, have proposed that reduction of the disulfide bond between the two subunits of the Ebola glycoprotein complex, GP1 and GP2, “is a critical step in the entry of Ebola virus into cells” (Sanders Lab, url).

Ozone, in vitro, instantly oxidizes reduced sulfhydryls to disulfides as shown in the chemical reaction below:

SH+SH+O3>S-S+H2O+O2. While ozone itself lasts only microseconds in blood, the reaction of ozone and blood lipids leads to the production of more stable but still highly reactive oxygen species (such as peroxides), which would react similarly, and perhaps mimic the pro-oxidant mechanism of immune system defense.

Aerobic cells are designed for redox shuttling and oxidant stresses. Viruses lack repair capacity. Shut out of the cell (inactivated), they cause no damage, but remain antigenically intact for immune response. However, aerobic mammalian cells can and do quickly repair membrane oxidation effects. One repair pathway activates the hexose-monophosphate shunt, which, incidentally, produces 2,3 DGP.

Finally, intravenous oxygen gas has been administered in significant volumes for decades in Europe. Numerous papers have reported beneficial effects on several physiological parameters. Regelsberger observed a general improvement in oxygen availability, eosinophilia, which can be valued as an increase in undetermined cellular immunological resistance. “Furthermore, rheological qualities of the blood as well as diuresis are improved, the release of oxygen into the tissue is increased, and the blood pH is normalized” (Schmidt, 2002). Intravenous oxygen gas in human volunteers induces eosinophil generated 15-LOX-1, a powerful anti-inflammatory enzyme, believed to be a key factor in inflammatory modulating effects of IV oxygen gas (Chaitidis, et. al., 2004).

These considerations caused lead author Rowen to speculate that OT might be an ideal candidate to actually cure EVD (and other viruses). Rowen has been using OT to treat virus and bacterial infections (outpatient setting) since 1986. He recruited Dr. Howard Robins, who refined a very inexpensive and relatively medical waste free technique of ozone administration- direct intravenous gas administration (DIV). DIV administers an oxygen/ozone gas mixture intravenously using a 27g winged infusion set. The clinician sets an ozone concentration up to 55 mcg/cc. (Approximately 98% O2 and 2% O3). Great care is taken to prevent “air”, which is 79% nitrogen gas and can cause an embolus, from entering the body. There are no reports in the worldwide literature, even after decades of use, that intravenous oxygen gas causes embolism. Desaturated venous blood is “thirsty” for it.

A protocol (Rowen-Robins Method) for EVD was developed inclusive of oral vitamin C, and a supplement supporting recycling of glutathione (Thiodox®, Allergy Research Group).

Figure 1: Robins demonstrating DIV technique on Rowen before gathering of SL professionals. Sixty cc of gas is administered in this treatment.

Rowen and Carew visited the Freetown Hastings Ebola Center and taught the method on 10/24/2014. The staff acknowledged a current 60% EVD mortality rate, considered, among the best results in Sierra Leone. For unknown reasons Ministry of Health authorities suddenly forbade the administration of OT on patients at the center while the educational session was in progress. Since all confirmed cases of Ebola suffered mandatory quarantine, it became extremely difficult to locate and treat cases outside this mandate. However, four cases did arise from within the facility amongst front line providers. These managed to receive OT. We now report these four case results. The first case, who was not tested, had had an accidental needle prick from Ebola contaminated blood three days earlier. All patients executed informed consents.

Materials and Methods

“DIV” ozone indicates direct intravenous ozone gas at 55 mcg/cc at a volume between 20-40 cc. “Rectal ozone” indicates ozone gas administered rectally at a concentration of 36 mcg/cc and volume between 150-350 cc. “Ozone water” was made by bubbling ozone gas at approximately 70 mcg/cc into water for 15 minutes. Administration volume was 300-500 cc, and administered orally. All cases were provided nutritional supplements Thiodox® and Buffered Vitamin C® (donated by Allergy Research Group). Thiodox dose: one twice daily. Vitamin C: four to eight grams daily during the days of ozone treatment.

Case Reports

Case 1 – Physician SK, 28, male, at the Hastings Ebola Center in Freetown jabbed himself with a contaminated needle. He was fearful to get an Ebola test, knowing if positive he would have been forcibly picked up and placed in quarantine and denied OT, which he feared would cost him his life. He was a physician Rowen and Carew had trained in OT at Hastings. He received 20 cc of DIV on October 23, 2014 as part of the training. The following is his verbatim and signed report edited only to remove names. What appears in brackets is editing by the authors for accuracy.
14th November: Needle prick in the red zone while trying to cannulate an EVD positive patient. This patient was in the recovery ward with no complaint and symptoms. She had done the blood (test) but it came positive and was waiting for the second specimen to be taken.
The needle went through the PPE and pricked me a finger length anteriorly above the wrist. I was wearing Tybek (the thinnest PPE). The prick happened just above the margin of the gloves, making me more exposed. Had it gone through the gloves, as in the case of another doctor, it wouldn't have penetrated the skin.
15th November: No symptoms, but planning to start OT.
16th November: No symptoms- feeling fine. Called Dr. Carew and went to see him. DIV [30 cc, 55 mcg/cc] done [one session]. [He also received 500 cc ozone water]. Was given Vit. C, Thiodox, colloidal silver. I was also given the ozone machine, couldn't use it - gas leak.
17th November: Fever at night, loss of appetite, bowel movements (unusual) and urge to empty my bowel. The urge was very strong. I tried to suppress it. I took ciprofloxacin, paracetamol, doxycycline, drank ORS. Couldn't sleep because of fever and the urge. I went after midnight to pass stools and I felt some comfort. The stool was not watery but not too hard (it was very soft). I came and slept.
18th November: Loss of appetite in the morning and weakness. Slightly febrile, muscular pain and joint (suppressible). Went directly to Blue Shield and finally Dr. Carew came with Jeff [McNamara]. Before their arrival I was really weak and couldn't stand. For too long had to sit down. But after drinking the ozone water Jeff prepared I regained my strength and felt much better. I also tried the [ozone] fog. Started rectal ozone [36 mcg/cc].
19th November: Slight weakness. Appetite is much better. DIV done, started working again. Did rectal ozone.
20th November: For the first time I did rectal ozone 3 times a day [36 mcg/cc]. One DIV. Slight weakness. Slept well at night. My temperature was 37.1 degrees Celsius.
21st November: My appetite is improving. DIV and rectal [as before].
22nd November: DIV and rectal. No complaints. Prepared ozone water.
23rd November: No complaints. Ozone water but no DIV and rectal.
24th-29th November: Ozone water only.

SK fully recovered, resuming his duties within just days of commencement of therapy.

Case 2 - Physician MB, 35, male, had close personal contact (hug) with another physician after the latter initially tested negative for Ebola. However, within 2 days a repeat test (PCR) came positive on the other physician (who died shortly thereafter). MB subsequently developed typical Ebola symptoms (fever, abdominal pain, vomiting, appetite loss, and later diarrhea) within 3 days. He was placed in quarantine, but was offered and accepted OT administered by the recovered physician SK.

At the appearance of symptoms, he received 30 ml DIV. When PCR testing returned positive, he was given an additional 2 DIV administrations consisting of: 40 cc each, 12-16 hours apart, at 55 mcg/cc. He also drank 300-500 cc of ozonated water after the intravenous treatments. Within 4 days, all symptoms had cleared. A follow-up test for presence of Ebola virus was negative 2 weeks later. The government publicly announced this case as a complete Ebola recovery in a physician national, but did not mention he had received OT.

Case 3- SS, 25, male nursing student on the Ebola front line, who was present for the Rowen and Robins ozone training in October 2014. He documented exposure to Ebola via damaged protective gloves enabling an Ebola patient’s blood to come in contact with his skin. He also, without protective gear, cared for a friend, who later was confirmed to have EVD. On December 2, 2014, SS developed fever, malaise and headache. He received 2 DIV ozone gas injections of 20 and 30 cc respectively at 55 mcg/cc, and drank ozone water (100 cc) on 3 consecutive days. Upon testing positive for EVD, (PCR) he was taken to the Kerry Town Ebola treatment center where he was not permitted further OT. However, he had a complete and non-complicated recovery.

Case 4 - IB, 24, male aid. Working in “red zone”. While bathing an Ebola patient, on or about November 24, 2014, he accidentally splashed body fluid contaminated water that went through his facemask and entered his eyes and mouth. Within 5-7 days, he developed progressive symptoms of extreme fatigue, body pains and vomiting. He started on anti-malarials. The next day, he informed the facility physician who immediately administered DIV ozone, 40 mcg/cc, 40 ccs. Within a few hours of the gas injection, almost all symptoms subsided. He also received two rounds of ozonated water (as in case 2). However, by then, he was about symptom free. His Ebola test proved positive (PCR) and he was placed in the treatment unit and prohibited from further DIV ozone. He did receive their usual treatment protocol consisting of D5W, Ceftriaxon (IV), Metronidazole (oral), Immunoboost (oral vitamin). He had an uneventful stay within the Ebola containment unit and was discharged in the first week of December.

Case 5 - GK was the female companion of a 67-year-old Sierra Leone senior physician who died of EVD. She had intimate contact with him at the time of his falling ill. Authorities placed her under home military quarantine (armed military guards), preventing any entry and exit, inclusive of anyone who might bring her OT. She was in great fear for her life and very much aware of OT, having urged her partner to accept it before he died. In the middle of the night she scaled a razor wire fence shredding her skin and evaded the guards. She arrived at Dr. Carew’s “Blue Shield” facility where she received: one DIV ozone treatment, and daily rectal ozone and ozone water for ten days. She also received vitamin C and Thiodox® twice daily. No symptoms developed.

An ozone-fogging device was deployed at Dr. Carew’s Blue Shield facility for decontamination and protection of Dr. Carew and all exposed to patients who were treated there.


EVD has a progressively explosive downhill course from the time of symptom appearance. Typically, death occurs within a week or less in the majority of cases. In all our ozone treated cases, symptoms did not progress from the start of OT, and symptomatic patients were totally free of all symptoms, inclusive of fever, generally by day 3 of treatment.

Through December 2014, Sierra Leone lost 11 out of 13 national physicians diagnosed with confirmed or probable Ebola. Both survivors received OT and quickly recovered. Following Rowen-Robins’ mission, one senior physician was offered and refused OT. He received convalescent serum and was transferred to the USA and died only 2 days later. Another senior physician likewise refused OT opting for ZMapp. He died while it was thawing. (Source: public news wires). A third EBV positive physician, Hastings trained, requested OT. Authorities refused and quarantined him. He died within 3 days.

Both senior authors had expected rapid recovery with OT, but admittedly not this fast (within a few days and with limited treatments). Rapid recovery was expected because of the violent nature of EVD, and the known direct countering biological benefits/effects of OT. This merits further discussion.

Ebola induced pathology includes rapid cellular entry, an explosion of viral particles into circulation, and rapid cellular re-entry perpetuating the cycle viciously. Then the repressed immune system “awakens” and pulls out all its weapons to do battle. But unfortunately, that battle results in a cytokine storm, wherein the immune system does more damage to the vascular system and tissues than does the virus. Death occurs due to capillary leaks, hemorrhage and organ failure.

Circulatory compromise – The final common denominator in any vascular insult is oxygen deprivation and resultant cellular injury and death. OT is known to improve rheological properties of blood, increase 2,3 DGP, shifting the oxyhemoglobin curve to the right and releasing more oxygen in tissues. Bocci, Menendez, and others have well demonstrated that OT enhances oxygen delivery and utilization. Ozone itself is oxygen. Clearly, in advanced EVD with vascular damage, tissues are starved of oxygen and energy production. Any oxygen delivery enhancement could potentially salvage cells that might otherwise die. Viral entry – The need for reduced sulfhydryl groups to enter cells, may be the Achilles heel for reversing the lethality of EVD (and other viruses). Sulfhydryl groups are key to activity of many cellular enzymes; aerobic cells may control enzyme activity by redox, providing means to activate/inactivate these enzymes. It appears from our cases EBV has a very narrow redox window, and that its envelope glycoproteins must be reduced as literature suggests. The symptomatic patients began a recovery essentially with the first oxidation (ozone) treatment. The senior authors did expect recovery, but within 5-7 days, considering the natural course of EVD. These symptomatic cases improved with the very first treatment. An oxidant stress to the blood stream carrying newly emerging viruses may be capable of oxidizing and inactivating sulfhydryl entry mechanisms. Such inactivated viruses will be incapable of entering the cell for further replication, while able to encourage healthy immune responses. Gonzales et. al., (2014) reported on a case of another vicious virus now likely endemic in the USA. A man (54) developed high fever and severe arthralgia among other symptoms. He was positive for Chikungunya virus. After receiving two intravenous infusions of vitamin C, 100 grams each, he immediately began recovery and was clear of symptoms in two days. A second paper reported combined ascorbate and intravenous hydrogen peroxide on symptomatic cases of Chikungunya virus observing fast symptomatic relief (Marcial-Vega, et. al., 2015). This parallels our observations with ozone. Importantly, ascorbate in these doses has been found to undergo a newly discovered metabolism. The lab of Mark Levine at the National Cancer Institute research reported a heretofore-unknown effect of high levels of plasma ascorbate. “Pharmacologic ascorbate can act as a pro-drug for H2O2 formation, which can lead to extracellular fluid [accumulation of H2O2]” (Chen, et. al., 2005). Bocci, et. al., (1998) theorized that the key mediator in ozone’s beneficial effects is H2O2. Oxidation therapy was reported as far back as 1920 to dramatically cut the mortality rate of influenza pneumonia in the great epidemic of that time. British physician Oliver (1920), in India, took only hopeless cases and nearly halved the death rate with intravenous H2O2 therapy. In the 1940’s, ozone’s sister therapy, ultraviolet blood irradiation therapy, was used to rapidly resolve viral influenza (Miley, 1942) and polio (Miley and Christensen, 1944). We have nothing, even in today’s modern world, which compares.

Cytokine storm - Ebola induces a cytokine storm. OT has been shown to significantly modulate TNF-alpha and inflammatory cytokines. Bocci has investigated and reported ozone as a cytokine inducer (Bocci, et. al., 1993). Bocci (personal communication to Rowen) called ozone the “ideal cytokine inducer”, inclusive of anti-inflammatory cytokines. EVD instigates pathologically high levels of NO. Ozone modulates NO (Bocci, et. al., 2007).

The actual extraordinary rapid recovery of the treated patients suggests that all three mechanisms may be at play, particularly viral inactivation. Ozone easily oxidizes SH groups to S-S groups, which, according to literature, is expected to inactivate viral entry. Ozonides, reactive oxygen species generated by OT such as peroxide species, would also easily oxidize reduced sulfhydryl groups based on simple chemistry. Our experience suggests that EVD has an extremely narrow redox window of infectivity. Even ozone exposures (rectal and water), far less powerful than DIV, appear to have assisted in dispatching symptoms and aiding recovery. We believe that the temporary oxidant stress to EVD patients oxidizes viral surface glycoproteins. The virus particles are unable to recover since they lack means to self-repair damage to their glycoprotein spikes. Also, the temporary oxidative stress created by ozone treatment stimulates the immune system to respond in more favorable conditions to the Ebola virus.

Additional damage to viral infectivity could be inflicted on the lipid envelope. The virus incorporates lipids from our own cell membranes. Infectivity is dependent upon a functional lipid membrane. Agents that attack the lipid envelope may be useful as antiviral drugs (Lorizate, 2011). Ozone directly attacks unsaturated fatty acids, which would be expected to be part of the Ebola lipid envelope. Aerobic cells repair such alterations. Viruses cannot. Lorizate lamented that compounds which could attack viral lipids lack specificity and are “thus unacceptably toxic.” OT, in use for decades, has no reported toxicity and may serve as an ideal lipid altering anti-viral agent.

Statistical probability: With Hastings center’s survival rate of only 40%, the statistical probability of 100% recovery arising from mere chance is 0.4(4), or 0.026%. Furthermore, lack of disease progression upon therapy commencement greatly magnifies the significance.

Ethics: International agencies, inclusive of the WHO (2014), called for the use of any reasonable treatment in the fight against Ebola. (“…the panel reached consensus that it is ethical to offer unproven interventions with as yet unknown efficacy and adverse effects, as potential treatment or prevention.”) Considering the high mortality of EVD, we then believe it unethical to withhold a known, decades old, safe therapy from EVD patients, which has a 60% probability of death, to do a double blind study, or to deny OT for prophylaxis. Effectiveness will be self-evident.

Cost: The ozone cost of treatment per patient was less than 10 USD excluding cost of ozone generator. Medical waste was limited to one 27 gauge “butterfly” needle per treatment (0.75 USD) and one syringe (reusable as ozone sterilizes the syringe as it fills each time) for each patient. Beyond modest cost of a reliable generator (1700 USD or less), the cost of DIV ozone will largely rest in labor costs.

Safety: The world literature is devoid of medical ozone toxicity reports when administered within the guidelines of the Robins DIV method or the more common method of major autohemotherapy. In the latter, between 50-200 cc of blood is removed, treated with ozone, and returned to the patient. During training, we treated several score Sierra Leoneans with DIV ozone without any toxicity except rare vein irritation. Both senior authors have performed thousands of ozone treatments with negligible untoward effects. Both have observed repeatedly rapid resolutions of both viral and bacterial infections in hours to days.

Availability: OT is not patentable; therefore it will fail to generate a profit for any developer or promoter. Hence, OT, though widely practiced, is not industry or mainstream promoted, and remains relatively unknown. This was a major reason Rowen and Robins chose to travel at their own risk and cost. By achieving success for the most dread virus on the planet, OT might attain its rightful place in healing and saving lives, regardless of lack of profit potential and industry glamour.

Weaknesses of this report: We acknowledge that these cases were treated early (soon after symptoms developed). None were critically ill. Ozone benefits on late stage EVD remain unknown.


Ozone therapy, a modality not well known or understood by conventional Western medicine, has performed as a safe and ideal therapy for EVD in all infected patients receiving it. EVD symptoms cleared within 2-4 days in all (four) symptomatic cases involving front-line health workers. No symptoms developed in a fifth extremely high-risk person. In contrast, two leading Sierra Leone physicians who contracted EVD and refused treatment both died, and one who requested, but was denied ozone treatment, also died, within weeks after our visit. DIV ozone is inexpensive, safe and leaves virtually no contamination. International organizations and governments would do well to immediately conduct a formal trial of this unique therapy for EVD, which could provide significant security, both for EVD prevention and treatment, and for dangerous font-line work.

Dedicated to: Terri Su (Rowen), MD and Linette Robins, whose unselfish love and bravery sustained the mission’s hardships. And, to the staff at Blue Shield facility who were willing to place themselves in harm’s way for a greater good. And, to all the people of Sierra Leone who have endured unspeakable tragedy and suffering which continues even to this day of submission.

Funding: Funding for supplies, equipment and airfreight came from private donors (see acknowledgments). No funding was provided for any of the authors. Rowen and Robins made the trip to Sierra Leone at their own personal expense.

Acknowledgments: The authors wish to express gratitude to Longevity Resources, Inc of Vancouver B.C. for generously donating 10 ozone generators for use in Sierra Leone, and to Allergy Research Group of California for its generous donation of oral Buffered Vitamin C and Thiodox to complement the ozone therapy. ACS 200 silver was supplied courtesy of Results RNA Company. Additional thanks go to the many selfless contributors and supporters of ozone therapy who spontaneously and generously donated unsolicited funds for the purchase of medical supplies and transport of supplies. Rowen and Robins received no financial support and personally paid all travel expenses out of their own funds. None of the authors have any financial disclosures to make.

Special thanks to ozone technician Jeff McNamara of Colorado, USA for introducing and bringing the parties together at this critical time and providing “ozone fogging” technology for front line protection.

Author contributions:

Dr. Rowen sourced the scientific references, drafted the manuscript, and compiled the transmitted information from Sierra Leone.

Dr. Robins provided training, education and expertise in his method of ozone delivery and traveled together with Dr. Rowen to Sierra Leone.
Dr. Carew coordinated all training in Sierra Leone, both didactic and practical, bringing in scores of health care professionals.
Dr. Kamara coordinated difficult retrieval of information and results back from Sierra Leone. Additionally, he visited each of the named patients and subsequently deceased physicians at his own peril to offer the therapy and obtain informed consent.
Dr. Jalloh was the supervisory physician to the treatment of case 2 and assisted with training at the Sierra Leone Ebola center


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PostPosted: Sat Jun 09, 2018 8:28 pm 
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I will chime in here on DIV and phlebitis.

I do limited DIV, and for the reason of phlebitis and losing veins. I do think DIV has great value.. I have heard from many patients that they get MUCH better results from DIV than from standard gravity MAH, from standard therapy trained physicians.
I began ozone with DIV in 1986 at 20 gamma. Over the years I have raised it to 30 gamma. I’ve seen very little problems at this level.
I have done these on myself, including just this week, when I accidentally stuck myself with a needle (rare but happening event for us all). I immediately went to machine and took 35 cc ozone gas at 33 gamma. No vein issues. In Sierra Leone, I took daily ozone at 55 gamma. My veins did not like it but came out ok.

Many, many years ago, in an experiment on myself before I knew anything or anyone else in the ozone world, I took ozone at 70 gamma and dearly acutely damaged my hand veins.. They appeared to be thrombosed. Not something I’ll ever do again. I immediately used castor oil packs, vitamin C and Wobenzym in large doses.. I was quite worried. By the third day, I could see blood running through my hand veins again and after some more time, the veins returned completely.... Now you would never know that I clotted off some large hand veins, they are just fine.

After that incident, I limited my gamma concentration for this procedure to 30.. I believe Howard uses higher amounts to get a faster response. He has to juggle cost, frequency and effectiveness. I weighed out on the side of the veins and was content to use less ozone. However, I largely dispensed with DIV (not completely for certain conditions) after I mastered HBO3 as the results we got were so superior to anything else I’d ever seen.

If bad vein access, DIV makes sense, but over time, if you keep hitting the same vein with lower gamma, you’ll possibly knock it off. And, you ABSOLUTELY have to know what your machine is producing by your own analysis. And know that the machine might change over time so it needs to be rechecked. I can usually tell by machine behavior if something is off, regardless of the machine make. But most people will not know if a machine has gone south. With my analyses, when I give myself 30 gamma, I know that I am giving myself 30 gamma.

So, I hope this helps the discussion. I have found castor oil packs very useful in vein phlebitis, which, incidentally, can occur from MAH and HBO3 but is much rarer.

Our workshop is this weekend and we will be discussing this. All seats are now gone. Our group will have a real treat though. A man from Guinea Africa, who lost friends in the Ebola epidemic, came all this way for me to treat his prostate.. His daughter came from east coast for horrible neuropathy and motor dysfunction in sciatic. I’ve made a video of her and it is posted on my youtube channel. They both will be there to share with the group the one shot cure for many many years of sensory and motor sciatic neuropathy, and that in just one week, his urinations have gone from 6 per night to 3 per night and improving. We will get the benefit of hearing how the man’s wife, a Polish nurse, who knew nothing about ozone therapy, learned what she could from my videos, and began using ear insufflation and ozone water to treat malaria, infection and even AIDS in Guinea. And, she is curing them!!!!!!! Including an AIDS case.

I may have to to just to check this out in Guinea. I have an invitation now.

Best regards,
Robert Rowen MD.

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PostPosted: Fri Jun 29, 2018 4:42 pm 
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Note: The accurate concentration of ozone is dependent on two things, oxygen flow controlled by a regulator and the accuracy of the ozone generator. As far as the generator, one has to rely on the company specs which should be checked on an individual basis from time to time.

The cheap pediatric oxygen regulators are notoriously inaccurate, even out of the box new. One should always check your individual oxygen regulator with a tub of water and a submerged graduated cylinder, plus a stop watch. For instant my regulator tested as following:

1/32 liters per minute reading = only provided 10 ml per minute when it should have did 31.25ml/min. Very slow and accordingly it would give a more concentrated ozone concentration than expected!

1/16 = 50 ml/min when it should have gave 62.5. Again, my regulator was giving a more concentrated reading than expected.

1/8 = 125 ml/min. Accurate!

1/4 = 264 ml/min. It was fast by 14 ml which would give a less concentrated ozone content than expected.

1/2 = did not test, but correct output would be 500 ml per minute

3/4 = did not test, but correct output would be 750 ml/ min.

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PostPosted: Fri Aug 03, 2018 4:45 pm 
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How does ozone really work molecularity from its inception to the tissues?

Medical ozone generators are fed with pure USP oxygen. The ozone generator then adds energy to the diatomic oxygen molecules via electrical discharges, hence breaking up the covalent bond between the oxygen atoms that form the molecule. This results in the O2 molecule dividing into two O1s. The O1 (usually referred to as oxygen radicals) tries to find a lower energy state and on the availability of other molecules, they will form 1 of 2 possible reactions with other oxygen atoms/molecules:

1) ozone (O1+O2=O3)
2) oxygen (O1+O1=O2)

Both these reactions will occur inside of an ozone generator, making the out-going gas from an ozone generator into a gas mixture between oxygen and ozone.

Once the ozone is produced it has to be injected and dissolved into the blood plasma or synovial fluid that needs to be treated. When the ozone is successfully dissolved, it will find components to react with. The ozone reaction or ozone oxidation occurs when the ozone is broken down into oxygen (O2) and oxygen radical (O1). The radical reacts with the components in the blood or synovial fluid. Once the oxygen radical has reacted with these components, all that remain is oxygen (O2) which in the case of blood plasma, should be readily absorbed by the RBCs.

Maximizing the ozone surface bubbles will only dissolve ozone until the saturation limit is reached. To dissolve more ozone, the saturation limit can be pushed further up by increasing the pressure. When dissolving ozone in plasma, the biggest challenge is to dissolve enough ozone to reach the beneficial curve of response. In the case of a relatively air-tight joint capsule, some pressure can be produced by syringe action.

Methods of increasing ozone dissolution in blood plasma can physically happen in one of two ways:

1) reduce the amount of oxygen that needs to be dissolved being fed into the ozone generator (resulting in higher ozone concentration)
2) increasing the pressure of the dissolution system (which happens in hyperbaric ozone therapy)

It all comes down to the physical law formulated by William Henry in 1803, also known as Henry’s law:

“At a constant temperature, the amount of a given gas that dissolves in a given type and volume of liquid is directly proportional to the partial pressure of that gas in equilibrium with that liquid.”

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PostPosted: Wed Aug 15, 2018 5:27 pm 
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The ozone destructor system attached to the ozone generator changes toxic ozone gas into oxygen and can easily be made as well from PCV pipe, silicone tubing, a few fittings, and a catalyst made of manganese dioxide and copper oxide. All of these parts and chemicals can be purchased on eBay. For a DIY ozone destructor:

1) 12" of PVC one inch tubing,
2) two end fittings for the PCV pipe that have threaded caps for easy removal, cleaning and recharging. One cap has the brass barb fitting attached and the opposite cap is drilled with ventilation holes,
3) a brass 1/8" air barb fitting and a three-way 1/8" barb fitting
4) a synthetic cotton type of material or loose weave cloth dusted with Manganese Dioxide (2/3) and Copper Oxide (1/3) and packed in the PCV pipe.

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PostPosted: Thu Apr 25, 2019 8:58 am 
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The major criticism of Ozone DIV from the medical establishment is the chance of air embolism occurring. Dr. Robins who has done over 280,000 procedures in his clinic writes this on the dangers of embolism:

I wish to clarify on oxygen and ozone gas embolism.

They are virtually impossible.

There has never been a published death from any form of DIV anywhere in the world, when performed by a licensed trained medical professional.

My clinic has now performed over 280,000 RMDIV's over the last 28+ years safely. New York State would not allow anything else.

Please also keep in mind that Dr.Bocci says that "ozone resolves instantly in blood".

The ISCO3 says in their position paper on ozone therapy:

“It is important to clarify that ozone as well as oxygen do not cause embolism, due to the fact that the blood is thirsty for them and dissolves them very quickly."

The main objection against all forms of DIV is the fear of “the danger of "air embolism". Robins Method of Direct Intravenous Ozone Therapy℠ (a.k.a. RMDIV℠) uses only medical grade oxygen and medical ozone generators with sealed systems, along with ozone resistant PTFE (teflon) and glass tubes.

If you look up “Air Embolism” in the medical literature you may be surprised. (Thank you again, Nick Fedan Phd.)

Most of the reports come from neurosurgery procedures where air often enters the venous system, and where 'clinical manifestations' of Vascular Air Embolism [VAE] appear in about 45% of the cases.

Anesthesiology references indicate those lethal doses of air in humans range from 200 to 500 cc of entrained vascular air, or 3 to 5 ml/kg.

That's a lot of air (well beyond RMDIV℠)at 4 ml/kg, which would mean 280 ml of air for a 154 lb person in a single dose. Not surprisingly, one report cites a fatal case of 100 ml of air per secondentering circulation during a subclavian venipuncture using a 14gauge (RMDIV℠ uses a 27gauge butterfly)needle. (Flanagan, Gradisar, Gross, and Kelly)

Also, it is not just the volume of air involved, but the speed of injection that also makes a difference (Toung, Roosberg, Hutchins).

In fact, an anesthesiology textbook indicates that "The Lethal Dose" [LD] in humans can be exceeded if the air is entrained slowly, allowing for hemodynamic compensation.

A bolus of air tends to lead to an increase in central venous pressure [CVP], a decrease in pulmonary artery pressure [PAP], and shock that is thought to be related to an air lock in the right ventricle.

On thecontrary, a slow infusion of air results in an increase in CVP/PAP, with compensatory increase in cardiac output" (Mongan).

Non-lethal doses of VAE are reported as 2ml/kg or less (Bricker).

In a 154 lb person, this would amount to 140 ml of AIR (not oxygen) entering the venous circulatory system. "The lungs appear to have a large capacity to compensate for air embolus within the pulmonary arterial circulation" (Emby and Ho).

Other reports indicate "clinical manifestations" of VAE start appearing with dosages of 100 ml or more, particularly in neurosurgery procedures where air often enters the venous system. (Mongan).

Anesthesiologists are trained to look out for these 'clinical manifestations', and to apply corrective measures. In fact, one of the measures taken is to have the patient breathe 100% oxygen.

The points gathered from these cursory reference examples are fairly straightforward:

--A VAE lethal dose is a lot of air in the venous system; it is around 280 ml in a 154 lb person.
--140 ml or less of air in the venous system is considered non-lethal in the literature.
--Non-lethal VAE cases are fairly common in neurosurgery, are expected, and anesthesiologists are trained to deal with them.

Given that the oxygen-depleted hemoglobin will absorb some of the gas, it would seem that staying below a single dose of 140 ml of oxygen/ozone would avoid even the "clinical manifestations" which the literature mentions, and which anesthesiologists must attend to in about 45% of neurosurgery cases.

Almost all RMDIV℠ treatments never exceed 115cc, but range between 5 and 115cc.

The RMDIV℠ protocol falls well within safe parameters reported in the medical literature.

Moreover, it is never AIR that is injected in the RMDIV℠procedure, but pure medical grade oxygen with a small percentage of ozone.

Therefore there is no danger of “air embolism” feared by other so called "ozone authorities" and ozone organizations.

In the future it would be in the best interest of the medical ozone world if all discussions of various forms of intravenous ozone therapy were kept in the area of scientific knowledge and not unsupported feelings and opinions.

Flanagan JP, Gradisar IA,Gross RJ, Kelly TR: "Air Embolus--a lethal complication of subclavian venipuncture -New England J Med, 1969 Aug 28;

Toung TJ, Rossberg, MI, Hutchins, GM: "Volume of Air in a Lethal Venous Air Embolism".- Anesthesiology, 2001 Feb; 94(2)

Mongan P. "A Practical Approach To Neuroanesthesia". - Lippincott, 2013

Bricker S: "The Anaesthesia Science"- Cambridge University Press, second edition, 2009

Emby DJ, Ho K:"Air embolus revisited-a diagnostic and interventional radiological perspective" SA Journal of Radiology. March 2006

Han D, Lee KS, Franquet T, et. al..: "Thrombotic and nonthrombotic pulmonary arterial embolism: spectrum of imagining findings - Radiographics. November 2003, Vol 23 (6)

O Donnell JM, Nacul, FE, Editors: "Surgical Intensive Care Medicine,second edition.. Springer, 2009

Muth CM, shank ES. Primary care: Gas embolism. New England J of Medicine. 2000;342:476–82

Fibel KH, Barness RP, Kinderknecht JJ: "Pressurized Intravenous Fluids Administration in the Professional Football Player. A Unique Setting for Venous Air Embolism".. Clin J Sport Med. 2015; 25(4):e67-9

Platz,E:"Tangential gunshot wound to the chest causing venous air embolism: a case report and review" - The Journal of Emergency Medicine.41 (2).2008

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PostPosted: Sun Jan 17, 2021 4:19 pm 
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A post of 1/17/2021 made by Dr. Howard Robins on vitamin C and ozone:

Hi everyone,
For whatever you may feel it’s worth:
All my patients have been taking oral vitamin c 5-6 times a day to stomach tolerance.
I have never had any oxidative stress reactions.
Virtually all my patients do extremely well using RMDIV and HBOT with or without Hemalumen.
Clearly the vitamin c does not stop the ozone from working.

As for IV vitamin c:
I began doing IV c in 1989. For over 6 years we were ordering 800 50ml vials from McGuff every two weeks.
Our average drip was 100gms. We also went as high as 200gms (200,000mgs a 5-7 hour drip).
Apparently anything over 50gms was meant to kill things while 50mgs or less was immune building.
After changing from MAH to DIV we did not use vitamin c to kill things only ozone.

We now do 25 or 50gm vitamin c infusions. We do them 30 minutes after RMDIV or as stand alone txs. We never do them on days when we do HBOT. Patients do not do oral vitamin c on days they do IV c.

Ozone and ozonides have so much to react with that we believe it meets minimal amounts of Vitamin c to be neutralized. Ozone seems to be used up within 10 minutes .

I get great results. This is all we do.

All doctors have opinions as do I. I base everything always on clinical observations and positive results.

We are all free do believe and do as we wish.

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